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WGW-2020

3.9 Pathophysiology (48)

Showing records 1 to 25

Display all abstracts in classification 3.9 Pathophysiology

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80079 Evidence of Hypoxic Glial Cells in a Model of Ocular Hypertension
Jassim AH
Investigative Ophthalmology and Visual Science 2019; 60: 1-15
79745 Reduced Cerebrospinal Fluid Inflow to the Optic Nerve in Glaucoma
Mathieu E
Investigative Ophthalmology and Visual Science 2018; 59: 5876-5884
79922 Demyelination precedes axonal loss in the transneuronal spread of human neurodegenerative disease
You Y
Brain 2019; 142: 426-442
79760
Kasi A
Neural Regeneration Research 2019; 14: 446-449
79984 Is primary open-angle glaucoma an ocular manifestation of systemic disease?
Wey S
Graefe's Archive for Clinical and Experimental Ophthalmology 2019; 257: 665-673
79762 Neuroinflammation as a target for glaucoma therapy
Adornetto A
Neural Regeneration Research 2019; 14: 391-394
79925 Blockade of microglial adenosine A receptor suppresses elevated pressure-induced inflammation, oxidative stress, and cell death in retinal cells
Aires ID
GLIA 2019; 67: 896-914
79932 Optic Nerve Head Astrocytes Display Axon-Dependent and -Independent Reactivity in Response to Acutely Elevated Intraocular Pressure
Tehrani S
Investigative Ophthalmology and Visual Science 2019; 60: 312-321
79425 All roads lead to glaucoma: Induced retinal injury cascades contribute to a common neurodegenerative outcome
Alqawlaq S
Experimental Eye Research 2019; 183: 88-97
79762 Neuroinflammation as a target for glaucoma therapy
Russo R
Neural Regeneration Research 2019; 14: 391-394
79745 Reduced Cerebrospinal Fluid Inflow to the Optic Nerve in Glaucoma
Gupta N
Investigative Ophthalmology and Visual Science 2018; 59: 5876-5884
79425 All roads lead to glaucoma: Induced retinal injury cascades contribute to a common neurodegenerative outcome
Flanagan JG
Experimental Eye Research 2019; 183: 88-97
80079 Evidence of Hypoxic Glial Cells in a Model of Ocular Hypertension
Inman DM
Investigative Ophthalmology and Visual Science 2019; 60: 1-15
79925 Blockade of microglial adenosine A receptor suppresses elevated pressure-induced inflammation, oxidative stress, and cell death in retinal cells
Boia R
GLIA 2019; 67: 896-914
79932 Optic Nerve Head Astrocytes Display Axon-Dependent and -Independent Reactivity in Response to Acutely Elevated Intraocular Pressure
Davis L
Investigative Ophthalmology and Visual Science 2019; 60: 312-321
79984 Is primary open-angle glaucoma an ocular manifestation of systemic disease?
Amanullah S
Graefe's Archive for Clinical and Experimental Ophthalmology 2019; 257: 665-673
79922 Demyelination precedes axonal loss in the transneuronal spread of human neurodegenerative disease
Joseph C
Brain 2019; 142: 426-442
79760
Faiq MA
Neural Regeneration Research 2019; 14: 446-449
79984 Is primary open-angle glaucoma an ocular manifestation of systemic disease?
Spaeth GL
Graefe's Archive for Clinical and Experimental Ophthalmology 2019; 257: 665-673
79760
Chan KC
Neural Regeneration Research 2019; 14: 446-449
79762 Neuroinflammation as a target for glaucoma therapy
Parisi V
Neural Regeneration Research 2019; 14: 391-394
79922 Demyelination precedes axonal loss in the transneuronal spread of human neurodegenerative disease
Wang C
Brain 2019; 142: 426-442
79745 Reduced Cerebrospinal Fluid Inflow to the Optic Nerve in Glaucoma
Paczka-Giorgi LA
Investigative Ophthalmology and Visual Science 2018; 59: 5876-5884
79925 Blockade of microglial adenosine A receptor suppresses elevated pressure-induced inflammation, oxidative stress, and cell death in retinal cells
Rodrigues-Neves AC
GLIA 2019; 67: 896-914
79425 All roads lead to glaucoma: Induced retinal injury cascades contribute to a common neurodegenerative outcome
Sivak JM
Experimental Eye Research 2019; 183: 88-97

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